Developmental changes in craniofacial morphology in subjects with Duchenne muscular dystrophy

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Developmental changes in craniofacial morphology in subjects with Duchenne muscular dystrophy.

Lateral cephalometric radiographs of 35 Japanese male patients suffering from Duchenne muscular dystrophy (DMD) were taken longitudinally from 10 to 20 years of age. Eighteen landmarks were placed and 15 angles and four linear distances calculated. Profile diagrams (profilograms) were produced to analyse changes in craniofacial morphological growth in the DMD subjects. The measurements were the...

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P164: Adeno-Associated Viral Vectors in Duchenne Muscular Dystrophy

Duchenne muscular dystrophy (BMD) is an inherited X-link disease. The incidence of this muscle-wasting disease is 1:5000 male live births. Mutation in the gene coding for dystrophin is the main cause of BMD. Most cases of this disease succumb to respiratory and cardiac failure in 3rd to 4th decades. The slow progression of BMD and recent achievement of gene therapies make it as an appropriate c...

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Detection of the Duplication in Exons 56-63 of Duchenne Muscular Dystrophy Patients with MLPA

Background Duchenne Muscular Dystrophy (DMD) is a deadly X-linked recessive disorder. This genetic disorder affects 1 among 3,500-5,000 males in the world. The majority of the patients are male, due to the type of inheritance. It affects most of the skeletal, the respiratory, and cardiac muscles, causing these vital organs to contract and eventually mortality.<br...

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Duchenne muscular dystrophy An overview of Duchenne muscular dystrophy

Duchenne muscular dystrophy (DMD) affects approximately 1 in 3,500 live male births [1]. It is caused by a large variety of mutations in the dystrophin gene. Because of these mutations, the body can no longer make dystrophin which is a protein important for stabilisation of the muscle cell during a contraction. Without dystrophin, muscle cells are damaged and slowly replaced by fat and scar tis...

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Noncompaction in Duchenne Muscular Dystrophy.

To the Editor With interest we read the article by Misumi et al. about a teenage boy with Duchenne muscular dystrophy (DMD) who had developed dilated cardiomyopathy, isolated left-ventricular hypertrabeculation / noncompaction (LVHT), systolic dysfunction, and fatal ventricular tachycardias (1). We have the following comments and concerns. In Fig. 2, the authors attempt to present LVHT on echoc...

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ژورنال

عنوان ژورنال: European Journal of Orthodontics

سال: 2005

ISSN: 1460-2210,0141-5387

DOI: 10.1093/ejo/cji074